What Is the Twisting of Blood Vessels to the Male Gonad?

The twisting of the blood vessels that supply the male gonad is a severe medical condition known as Testicular Torsion. This event occurs when the testicle rotates, causing the spermatic cord—the structure it is suspended from—to twist upon itself. The spermatic cord contains the blood vessels, nerves, and the duct that carries sperm. The twisting motion effectively cuts off the blood supply and compromises the testicle’s ability to receive oxygenated blood, classifying it as a urologic emergency that requires urgent intervention.

Understanding the Anatomy and Mechanism

Testicular torsion is fundamentally a mechanical problem involving the spermatic cord, which extends from the abdomen into the scrotum and contains the testicular artery and veins. When the testicle rotates, the cord twists, first obstructing the thinner veins that drain blood and then the thicker arteries that supply it. This obstruction leads to venous congestion and swelling, followed rapidly by ischemia, or the loss of blood flow and oxygen. Testicular tissue cannot survive long without oxygen.

The majority of cases, particularly in adolescents and young adults, are caused by a congenital anatomical variation called the “bell clapper” deformity. In males with this trait, the testicle is not properly anchored to the inner scrotal wall, allowing it to rotate freely within the scrotum. Torsion is most prevalent in adolescence, typically between the ages of 12 and 18, but it can occur at any age, including in newborns.

Immediate Signs and Symptoms

The most telling sign of testicular torsion is the sudden onset of severe, excruciating pain in one side of the scrotum. This pain usually appears without warning, often waking an individual from sleep. It does not subside with rest, positional changes, or standard pain medication. The severity of the pain is a direct result of the acute lack of blood flow.

Accompanying the sharp scrotal pain, many individuals experience abdominal pain, nausea, and vomiting. Upon physical examination, the affected testicle is often noticeably swollen and may be positioned higher than the unaffected testicle due to the shortening of the twisted spermatic cord. A key finding that distinguishes this condition is the absence of the cremasteric reflex, which is the normal upward movement of the testicle when the inner thigh is lightly stroked.

The constellation of these symptoms—especially sudden, intense pain combined with swelling and abnormal positioning—requires immediate evaluation in an emergency setting. Waiting to see if the pain resolves naturally is dangerous, as the viability of the testicle is directly tied to the speed of treatment. Even if pain briefly subsides, which can happen if the testicle temporarily untwists, a medical consultation is necessary due to the high risk of recurrence.

Time-Sensitive Treatment and Intervention

Testicular torsion is a time-dependent diagnosis, and intervention must be swift to prevent permanent loss of the testicle. While a physical examination is the initial step, an ultrasound utilizing color Doppler is often used to visualize blood flow within the spermatic cord and confirm the diagnosis. If clinical suspicion is high based on symptoms, the surgical team may proceed directly to the operating room without waiting for imaging to save time.

The definitive treatment is a surgical procedure known as orchiopexy, which begins with an incision in the scrotum to access the testicle. The surgeon first manually untwists the spermatic cord, a process called detorsion, to restore blood flow to the affected gonad. After detorsion, the testicle is secured to the inner wall of the scrotum with stitches to prevent future twisting. This procedure is also performed on the unaffected testicle, as the bell clapper deformity is often present on both sides.

The timing of this intervention is paramount, as the survival rate of the testicle declines sharply as the time without blood flow increases. The optimal window for treatment is within four to six hours of symptom onset, where the salvage rate is nearly 100%. If treatment is delayed past 12 hours, the chance of saving the testicle drops significantly, and after 24 hours, the salvage rate is very low. In immediate situations, a physician may attempt manual detorsion in the emergency room to temporarily restore blood flow, but surgery is still mandatory afterward to secure the testicles.

Potential Outcomes and Recovery

The outcome following testicular torsion is highly dependent on the duration of ischemia before blood flow is restored. If the testicle is successfully untwisted within the critical window, it is typically saved, and a full recovery is expected. If the tissue is determined to be non-viable due to irreversible damage, the surgeon must perform an orchiectomy, which is the surgical removal of the testicle.

Even when salvaged, there can be long-term effects, including a potential reduction in fertility. The loss of one testicle generally does not impair the ability to father children, as the remaining testicle compensates for hormone and sperm production. The orchiopexy procedure secures both the affected and non-affected testicles, virtually eliminating the risk of future torsion. Recovery typically involves a few days of pain management and requires avoiding strenuous activity for several weeks.