What Happens If You Can’t Burp?

The inability to burp, known medically as abelchia, is a physical dysfunction preventing the body from performing a necessary biological function: the expulsion of swallowed air and digestive gases. Burping, or eructation, is the primary way the body releases air that enters the digestive tract, often through swallowing air (aerophagia). When this release mechanism fails, the air and gas become trapped, leading to a distinct and often debilitating set of symptoms. This condition is formally known as Retrograde Cricopharyngeus Dysfunction (R-CPD).

The Symptoms of Trapped Air

Individuals who cannot burp experience a unique and distressing collection of symptoms resulting from the trapped air. The inability to release air from the chest and throat is the defining feature of the condition. This trapped gas creates a painful sensation of pressure that builds up beneath the sternum and in the lower neck area. The discomfort often worsens significantly after eating or drinking, particularly with carbonated beverages.

A frequent symptom is the presence of loud, involuntary gurgling noises originating from the throat or chest. These “croaking” sounds occur when the trapped air attempts to escape but is blocked, causing the gas to rumble and vibrate against the closed upper esophageal sphincter. Patients often report feeling the air move up their esophagus, only to be abruptly stopped and forced back down, intensifying the feeling of internal pressure. This audible symptom is often a source of social anxiety for those affected.

The Underlying Mechanism

The physiological cause of the inability to burp centers on the cricopharyngeus muscle, located in the throat. This muscle is part of the upper esophageal sphincter, acting as a valve at the top of the food pipe. Its normal function is to relax momentarily to allow food, liquid, and swallowed air to pass down into the esophagus when swallowing. Immediately afterward, it contracts tightly to prevent the contents of the esophagus from coming back up.

In individuals with R-CPD, the cricopharyngeus muscle functions correctly for the downward movement of food. However, it fails to relax and open when the body needs to expel air upward from the esophagus or stomach (retrograde movement). The muscle remains contracted, preventing the necessary venting of gas. The underlying cause of this muscular dysfunction is often idiopathic, meaning it arises spontaneously without a clearly identifiable trigger.

Immediate and Chronic Physical Effects

When the cricopharyngeus muscle prevents the upward release of gas, the trapped air must find an alternative route through the digestive system. The air travels downward into the stomach and intestines, resulting in severe abdominal bloating and distension. This buildup of gas causes the abdomen to swell noticeably, often intensifying throughout the day. This distension is frequently accompanied by abdominal pain and cramping as the gas creates significant internal pressure.

The trapped gas eventually exits the body as excessive and frequent flatulence, which is the body’s only remaining mechanism for venting the air. This increased flatulence can dramatically affect a person’s quality of life and social interactions. In some cases, the severe abdominal distension can push upward on the diaphragm, leading to a sensation of shortness of breath. The combination of physical symptoms and social discomfort contributes to increased anxiety and a tendency to avoid situations involving eating or drinking.

Diagnosis and Treatment Options

Diagnosis of R-CPD is primarily clinical, meaning a specialist bases the determination on a detailed review of the patient’s specific symptoms and history. There are no standardized diagnostic tests for the condition, so a physician, often an Ear, Nose, and Throat (ENT) specialist or a gastroenterologist, must first rule out other conditions. The inability to burp since childhood, combined with the characteristic gurgling and bloating, is highly suggestive of R-CPD.

The most effective and widely accepted treatment involves a targeted injection of Botulinum Toxin (Botox) into the cricopharyngeus muscle. Botox is a neurotoxin that works by temporarily weakening or paralyzing the injected muscle. The goal of the injection is to force the muscle to relax, allowing air to pass upward. This procedure is often performed under general or local anesthesia, helping the patient learn how to burp while the muscle is temporarily relaxed. A single injection has a high success rate, with studies showing that approximately 80 to 90 percent of patients gain the ability to burp, often leading to a lasting resolution.