What Are the Symptoms of L5-S1 Nerve Damage?

The symptoms of nerve damage originating from the lower lumbar and sacral spine are distinct, reflecting the function of the individual nerve roots involved. The L5 and S1 nerve roots are the lowest pair of lumbar and the highest pair of sacral nerves. Compression or irritation of these roots is a common cause of lower body symptoms. Since these two nerve roots form a major part of the sciatic nerve, damage often results in radiating leg pain known as sciatica. The L5-S1 segment is a frequent site of injury because it handles significant biomechanical stress, transferring the load from the upper body into the pelvis and legs.

Sensory Symptoms: Pain and Numbness Pattern

Nerve root compression causes sensory symptoms that follow a predictable pattern known as a dermatome, a specific area of skin innervated by a single spinal nerve. Pain and numbness along the L5 nerve root typically trace a path down the side of the thigh and calf. This sensory disturbance continues across the top (dorsum) of the foot and often extends specifically into the big toe and the space between the first and second toes. The sensation is commonly described as a sharp, burning pain, tingling, or a feeling of “pins and needles” along this distribution.

Sensory symptoms related to the S1 nerve root follow a different, posterior path down the leg. Pain and numbness for S1 radiculopathy radiate down the back of the thigh and calf. This pattern then travels to the outer edge and sole of the foot, often involving the pinky toe and adjacent toes. Although L5 and S1 symptoms can overlap, mapping the precise location of sensory loss helps locate the specific nerve root under pressure.

Motor Symptoms: Weakness and Muscle Control

Motor symptoms are characterized by weakness in the specific muscle groups, or myotomes, controlled by the affected nerve root, leading to functional deficits. Damage to the L5 nerve root primarily weakens the muscles responsible for ankle dorsiflexion, which is the action of lifting the front of the foot toward the shin. Patients may also experience a pronounced weakness in the extensor hallucis longus muscle, making it difficult to lift the big toe upward. The resulting inability to lift the foot during walking is a classic sign of L5 nerve damage known as “foot drop.”

Foot drop causes the foot to drag on the ground while walking. L5 nerve compression can also affect the gluteus medius muscle, which helps stabilize the hip and pelvis during walking. Weakness from S1 nerve damage affects the opposite movement, primarily impacting ankle plantarflexion, the ability to point the foot and toes downward. This weakness makes it difficult to stand or walk on tiptoes because the calf muscles are compromised. S1 damage may also decrease the strength of the hamstring muscles, which are needed for knee flexion.

Reflex Changes and Gait Impact

L5-S1 nerve damage impacts the body’s involuntary reflexes, which are a key part of a neurological examination. The S1 nerve root is responsible for the integrity of the Achilles tendon reflex, sometimes called the ankle jerk reflex. When this reflex is tested, a diminished or completely absent response is a strong indicator of S1 nerve root compression. The L5 nerve root does not have a primary, easily tested deep tendon reflex associated with it, which distinguishes its symptoms from those of S1 damage.

The combination of sensory loss and motor weakness alters a person’s gait. L5-related foot drop often results in a compensatory “steppage gait,” where the leg is lifted excessively high to prevent the toes from catching on the ground. When the foot is set down, the lack of dorsiflexion control may cause a characteristic “slapping gait.” S1 weakness, due to the loss of calf muscle push-off strength, can make it difficult to climb stairs or walk up inclines.

When to Seek Emergency Medical Attention

While most L5-S1 symptoms are not life-threatening, certain severe signs indicate a medical emergency requiring immediate intervention. These “red flag” symptoms suggest significant compression of the cauda equina, the bundle of nerve roots at the base of the spinal cord. The most serious symptoms include sudden loss of bladder or bowel control, such as urinary retention or incontinence.

Any new or worsening numbness in the “saddle area,” which includes the groin, buttocks, and genital region, is also a sign of severe compression. This specific pattern of sensory loss is known as saddle anesthesia. Rapidly progressing weakness in both legs is another indication that the pressure on the nerves is severe and widespread.

If any of these symptoms appear, particularly in combination with lower back pain, it is necessary to seek emergency medical attention. Prompt surgical decompression in these cases can be crucial to prevent permanent neurological damage.